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      "body": "@@ -202,23 +202,119 @@\n 10.png) \n-diventa\n+grazie alla continua ricerca del Dr. Caletti, Medico Choirurgo Nutrizionista, diventa ancora pi%C3%B9 Super:\n  %0A!%5BFlac\n@@ -13790,22 +13790,32 @@\n nti nel \n-Res\n+**LipoiCo  \n Q10\n+**\n  sono si\n@@ -21268,21 +21268,27 @@\n er%5B31%5D.%0A\n+%0A\n **\n-Res\n+LipoiCo \n Q10**%0ANa\n",
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      "body": "Congratulations @chefren! You have completed some achievement on Steemit and have been rewarded with new badge(s) :\n\n[![](https://steemitimages.com/70x80/http://steemitboard.com/notifications/voted.png)](http://steemitboard.com/@chefren) Award for the number of upvotes received\n\n<sub>_Click on the badge to view your Board of Honor._</sub>\n<sub>_If you no longer want to receive notifications, reply to this comment with the word_ `STOP`</sub>\n\n\nTo support your work, I also upvoted your post!\n\n\n**Do not miss the [last post](https://steemit.com/steemitboard/@steemitboard/steemitboard-world-cup-contest-spain-vs-russia) from @steemitboard!**\n\n---\n**Participate in the [SteemitBoard World Cup Contest](https://steemit.com/steemitboard/@steemitboard/steemitboard-world-cup-contest-collect-badges-and-win-free-sbd)!**\nCollect World Cup badges and win free SBD\nSupport the Gold Sponsors of the contest: [@good-karma](https://v2.steemconnect.com/sign/account-witness-vote?witness=good-karma&approve=1) and [@lukestokes](https://v2.steemconnect.com/sign/account-witness-vote?witness=lukestokes.mhth&approve=1)\n\n---\n\n> Do you like [SteemitBoard's project](https://steemit.com/@steemitboard)? Then **[Vote for its witness](https://v2.steemconnect.com/sign/account-witness-vote?witness=steemitboard&approve=1)** and **get one more award**!",
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      "permlink": "acidi-grassi-polinsaturi-omega-3-epa-da-olio-di-krill-protettivi-per-il-diabete-1",
      "title": "ACIDI GRASSI POLINSATURI OMEGA-3 (EPA da Olio di Krill) PROTETTIVI PER IL DIABETE-1",
      "body": "L’innesco del processo autoimmunitario che ha come conseguenza la distruzione (spesso solamente parziale) delle cellule beta del pancreas (affezione definita “diabete di tipo 1”), non  è  ancora  ben  chiarito.  Una  multifattorialità  è il più  probabile  dei  meccanismi patogenetici.  Sappiamo  però  che  i  fattori  alimentari  (le lectine come  il  glutine per esempio) (1),  c’entrano  non  poco  in  questo  meccanismo il  che  implica  che  il  sistema immunitario presente  nell’intestino (SNE)  sia  implicato  nella  patogenesi  della  malattia (2). Uno stato infiammatorio è presente nell’intestino strutturalmente sano dei diabetici di tipo 1 in età pediatrica (3) e l’alterata permeabilità intestinale che accomuna questi pazienti depone per l’origine intestinale e infiammatoria della malattia (4).\n\n![a51aa80a606b6d7f254abe4802179ae3.png](https://cdn.steemitimages.com/DQmWu9Tqn7fZAc1M7V5EpkX2pARYdqLderjFUPgAmVeaYbq/a51aa80a606b6d7f254abe4802179ae3.png)\n\nQueste premesse offrono dunque un  razionale  di  utilizzo degli  acidi  grassi  Omega-3(EPA), dato  il  loro  potente effetto antinfiammatorio,  \n\n![krill gold.jpg](https://cdn.steemitimages.com/DQmPFBrvT1X78PLNJKxWmGHyECtEFE6S9RDMC9K4GJWjz1J/krill%20gold.jpg)\n\ncome  mezzo  protettivo nei  confronti  della  distruzione  delle  cellule pancreatiche produttrici di insulina purché le    supplementazioni inizino molto precocemente rispetto all’insorgenza della malattia. Le  membrane  cellulari  sono  la  sede dove gli acidi grassi polinsaturi come l’EPA (Acido  Eicosanpentaenoco,  un  Omega-3), l’AA (acido Arachidonico, un Omega-6) e il DGLA (Acido DihomoGammaLinoleico) si depositano  per  divenire  precursori  di  citochine  dette  “pro-infiammatorie” o “antinfiammatorie. Queste citochine determinano in ultima analisi proprio  la  capacità  di funzionamento  delle  stesse  cellule  da  cui  derivano ed  evidentemente è lo  spostamento quantitativo verso uno o l’altro di questi Acidi grassi polinsaturi ad indirizzare le cellule verso un funzionamento corretto o scorretto in risposta agli stimoli (infiammazione Acuta Fisiologica) oppure verso il funzionamento alterato (Infiammazione Cronica Patologica). E’ dunque una questione di numeri,  dato  che  sela  quantità  di  EPA,  AA  e  DGLA nello spazio di membrana è adeguata, in  quell’organo o organismo nessuna  patologia  cronica potrà  mai  innescarsi.  Ma  se  al  contrario  la  quantità  di  AA  man  mano  aumentano  (le membrane   cellulari   sono   in   continuo   rimaneggiamento   e   la   loro   qualità   **dipende giornalmente dal nostro comportamento alimentare**), il destino di quella cellula, da un certo  momento  in  poi  e  con  progressivo  peggioramento,  sarà  quello  di  sviluppare comportamenti infiammatori cronici anche in totale assenza di stimoli lesivi. Viene dunque messo in moto un “viaggio” di tipo infiammatorio che sfocerà, anni dopo, nella patologia cronica infiammatoria. Questo succede dunque anche per patologie come il Diabete 1. È noto che prima dell’insorgenza della malattia conclamata, per mesi od anni appaiono nel sangue  gli  anticorpi  (IA) diretti  contro le  cellule  beta  del  pancreas  e  alcuni  studi  di  tipo epidemiologico collegano l’insorgenza di questa patologia al precoce consumo di frumento e derivati nei bambini (5). Dato chela produzione anticorpale (IA) segue un comando di tipo infiammatorio e che l’infiammazione  cellulare  cronicizzando può  diventare  più  o  meno violenta  e  distruggere  con  proporzionale  gravità  e  velocità  le  cellule  pancreatiche sintetizzanti l’insulina, è evidente che attraverso la gestione dell’infiammazione metabolica si potrebbe rendere l’aggressione autoanticorpale meno intensa e la evoluzione clinica del paziente meno aggressiva. Dato che la carenza di EPA o all’opposto un aumento di AA cellulare portano ad un incremento di infiammazione, ne consegue che uno squilibrio di queste molecole sia associabile ad un aumentato rischio di malattie autoimmunitarie (come appunto il  Diabete  di  tipo  1)  in  soggetti  predisposti. A  conferma  di  ciò,  uno studio prospettico che ha coinvolto 1770 bambini di 6,2 anni di età media, nel Colorado (USA) tra il Gennaio 1994 e Novembre 2006 ha evidenziato che l’assunzione dietetica di Acidi grassi Omega-3 durante l’infanzia è associata alla diminuzione del rischio di ammalarsi di Diabete di tipo 1 in soggetti a rischio di malattia (6). A questo punto la domanda è: siccome la  carenza  dietetica  di  EPA  aumenta  infiammazione  e  dunque  rischio  di  ammalarsi  di Diabete di tipo 1 e la supplementazione migliora l’evoluzione della patologia in chi già ha la  malattia  e  riduce  il  rischio  in  chi  ancora  non  ce  l’ha, come  mai  nessun  Medico diabetologo  oltre  al  sottoscritto  (che  Diabetologo  non  è)  prescrive  olio  di  krill  (la  fonte migliore di EPA) ai pazienti con diabete di tipo 1? \n\nAncora una prova, dunque, per favorire e non vietare l’assunzione in gravidanza e nei bambini piccoli di Omega-3 e precisamente EPA (e DHA) tramite **Olio di Krill**.\n\nQui potete leggere la scheda del prodotto migliore sul mercato mondiale che ho io stesso scelto per me e i miei pazienti: \nhttps://steemit.com/ita-health/@chefren/krill-oil-gold-qualitativamente-eccezionale-antinfiammatorio-per-eccellenza-interviene-in-modo-estremamente-benefico-sulla\n\n**Riferimenti**\n\n1. RenataAuricchioetalii,In Vitro–Deranged Intestinal Immune Response to Gliadin in Type 1 Diabetes in DiabetesJul2004,53(7)1680-1683\n2. Vaarala O: The gut immune system and type 1 diabetes. AnnNYAcadSci958:39–46,2002\n3. Westerholm-Ormio  M,  Vaarala  O,  Pihkala  P,  Ilonen  J,  Savilahti  E:  Immunologic  activity  in  the  small intestinal mucosa of pediatric patientswith type 1 diabetes. Diabetes52:2287–2295,2003\n4. Carratù R, Secondulfo M, De Magistris L, Iafusco D, Urio A, Cardone MG, Pontoni G, Cartenì M, Prisco F:  Altered  intestinal  permeability  to  mannitol  in  diabetes  mellitus  type  1. JPediatrGastroenterolNutr28:264–269,1999\n5. Norris JM, Barriga K, Klingensmith G, Hoffman M, Eisenbarth GS, Erlich HA,Rewers M. Timing of initial cereal exposure in infancy and risk of isletautoimmunity. JAMA. 2003 Oct 1;290(13):1713-20.\n6. Jill  M  Norris  e  coll.  Omega-3  Polyunsatured  Fatty  Acid  Intake  and  Islet  Autoimmunity  in  Children  at Increased Risk for Type 1 Diabetes. JAMA 2007;298:1420-8.",
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      "body": "#\n# upvote for me please? https://steemit.com/news/@bible.com/6h36cq\n#",
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      "permlink": "vaccino-antinfluenzale-o-discarica-di-rifiuti-tossici",
      "title": "VACCINO ANTINFLUENZALE O DISCARICA DI RIFIUTI TOSSICI?",
      "body": "![02fb778f651f361766b010e70d7f303b.png](https://cdn.steemitimages.com/DQmPZQhAgtL77mLMcx98PBtAajrzMxhDyvxnYGpJBrdzZyh/02fb778f651f361766b010e70d7f303b.png)\n\nUno  studio  Slovacco (1) eseguito su ratti femmine ha rilevato che l’iniezione di Polisorbato  80,  conosciuto  anche come Tween 80, entro la prima settimana dalla nascita dei soggetti, causa lo sviluppo di danni alla vagina e  all'utero,  cambiamenti  ormonali,  deformità ovariche  e  infertilità.  Il  Polisorbato  80 è un  detergente (surfattante)  utilizzato  per  promuovere  il  superamento  della  barriera  emato-encefalica  di  farmaci  o vaccini. I vaccini antinfluenzali della Glaxo (Fluarix) e quello delle Novartis (Agriflu), il vaccino contro il Papilloma virus Gardasil e molti altri in commercio lo contengono. L’adiuvante MF59, anch’esso pericoloso,  formato  sia  da  squalene  sia  da  Polisorbato  80  viene  inserito  in  molti  altri  vaccini.  Come adiuvante  lo  squalene  viene  inserito  nei  vaccini  Europei  ma  non  in  quelli  americani  perché  non  è approvato dalla FDA. \n\n![c10e95095245a0b73071f9f68f3b9c9f.png](https://cdn.steemitimages.com/DQmTEMsN4N8VFB8StnuMZ3LVnGof9eS9DYmWmuWKuyRFerj/c10e95095245a0b73071f9f68f3b9c9f.png)\n\nCosa troviamo nel vaccino infleunzale Fluarix? (2) Formaldeide, un ben noto agente cancerogeno, il TritonX-100 (un  detergente),  idrocortisone  e  un  antibiotico:  la  gentamicina,  che  può  indurre  risposte allergiche in alcuni individui. Che questi adiuvanti tossici e dannosi per la fertilità dei soggetti trattati, ratti, sia da estendere anche agli esseri umani non è stato chiarito in quanto la Ricerca non si è prodigata molto  in  questa  direzione.. Nel  foglietto  illustrativo  del Fluarix,  per  esempio, viene  scritto  che  la possibilità  di  indurre  infertilità,  mutagenicità  o  carcinogeneticità  maschile  non  è  stata  testata  negli animali mentre la vaccinazione nelle femmine di ratto non ha effetti sulla fertilità. Grazie alle numerose leggi che tutelano i produttori di vaccini promulgate dai vari Governi nel mondo, non sarà mai possibile ritenere  la GlaxoSmithKline o  qualunque  altra  azienda  farmaceutica responsabile di  una  eventuale sterilità post vaccinale.\n\n**Riferimenti**\n\n1Gajdová M, Jakubovsky J, Války J. Delayed effects of neonatal exposure toTween80 on female reproductive organs in rats. Food Chem Toxicol. 1993Mar;31(3):183-90.2https://www.fda.gov/downloads/BiologicsBloodVaccines/Vaccines/ApprovedProducts/UCM220624.pdf",
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